Abstract

The development of a unique genetic sexing strain to control fruit fly populations has repetedly failed. But why do they fail to succed? We previously demonstrated in Certitis capitata (Wied.) that the autosexing mechanism must be developed on the germplasm of the population to be controled. The present integrative study addresses the causes for the lack of success, thus studying: 1- compatibility tests between Anastrepha fraterculus (Wied.) germplasms from different geographic origins; 2- the genotype by environment interaction component of phenotypic variation in A. fraterculus and 3- essential knowledge on polymorphisms of the Y-chromosome carrying the marker linked to male sex.

Keywords

molecular cytogenetics, genetic sexing, fruit flies, embryogenesis