Abstract

Experimental data are described, showing that glucocorticoid-induced growth retardation may have quite complex mechanisms in various organs and tissues, with age-dependent differences of these mechanisms. It is expected that these data may contribute to clarification of the role of glucocorticoids as mediators of programming / imprinting and embedding phenomena in DOHaD paradigm.

Keywords

glucocorticoids, growth, ontogeny, organs and tissues